To help you get started on your quest for healing, I have presented some basic questions that you may be asking yourself, and have linked some initial information to the question. (The question will be linked to the article by clicking over it. An excerpt from the article may follow each question or, I may just link the article to the question if a small excerpt does not address it satisfactorily.) After you find out what interests you, continue down the page to view a list of links provided by others from the message board to see if the topic you are interested in is reflected there. If not, you will likely have a good place to start after reading the article provided. It is important to remember that the research provided is not necessarily (nor likely) 100% accurate, and further research will be necessary to convince you of it’s validity. Also, thyroid research is moving quickly, therefore causing what seems like “good” information now, to become outdated and inaccurate over time. So, it is important to continue your research as new information is uncovered.
Patients with Hashimoto’s thyroiditis, an autoimmune disorder responsible for most instances of hypothyroidism, do not have constant levels of destructive autoantibodies. Nor do patients with Graves’ disease. Like most autoantibodies, these IgG immunoglobulins only last about 120 days. New antibodies are formed at a rate proportional to the immune system’s response. How can these patients be expected to thrive on one standard thyroid hormone replaement dose or one dose of anti-thyroid drugs while their immune status is ignored? Furthermore, dying thyroid cells, especially after radioiodine ablation, release thyroid hormone and autoantibodies. This causes transient symptoms of hyperthyroidism, although in some cases these symptoms may persist for longer than one year. No wonder many hypothyroid patients feel hyper one minute and hypo the next.
…having one autoimmune disease increases your risk slightly of developing another autoimmune condition.
Is it true that vaccinations may contribute to Hashimoto’s Disease? (Although this article reflects an animal study, I think it is a significant account of how it may effect humans)
….. In dogs as well as humans, the body sometimes forms antibodies against itself (self antigens) which can lead to diseases of the pancreas (diabetes), thyroid (Hashimoto’s Disease), collagen and fibronectin (Scleroderma, Lupus),cardiolipin (Cardiomyopathy), etc. The body literally attacks itself to cause the autoimmune disease.
The vaccinated group developed significant levels of autoantibodies against: fibronectin, laminin, DNA, albumin, Cytochrome C, transferrin, cardiolipin, collagen. The responses varied among individual animals, probably reflecting genetic differences. The clinical significance of those autoantibodies remains to be determined, but speculation must be that something in the vaccines is one of the etiologies (in the genetically susceptible dog) of such diseases as Cardiomyopathy, Lupus Erythematosus, Glomerulonephritis, etc. I apologize for using these technical terms but this is a complex study and adds validity to the report.
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